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Clinically, PM 10 particles likely provoke airway inflammation via the release of mediators that exacerbate lung disease in susceptible individuals 70

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Given the critical role played by NOX in AD, there is potential for selectively targeting specific NOX subtypes to rectify eNOS uncoupling and mitochondrial dysfunction, thereby offering a viable approach for the prevention and treatment of AD ( from the body, diminish the infiltration of inflammatory cells, and retard the aging process (185)